Neurological Disorders and Stroke

Neuroinflammation and Neurological Disorders: Mechanisms, Impact, and Therapeutic Strategies

Neuroinflammation, characterized by the activation of immune cells in the central nervous system, has emerged as a critical factor in the pathogenesis and progression of various neurological disorders. This session focuses on understanding the mechanisms of neuroinflammation, its impact on neurological disorders, and the development of therapeutic strategies to mitigate its effects.

During this session, leading experts in the field will present the latest research findings on neuroinflammation and its role in neurological disorders such as Alzheimer's disease, Parkinson's disease, multiple sclerosis, and stroke. They will delve into the underlying molecular and cellular mechanisms that drive neuroinflammation, including the involvement of immune cells, cytokines, and oxidative stress. Discussions will revolve around the impact of neuroinflammation on neuronal function, neurodegeneration, and cognitive decline. Presentations will highlight the potential therapeutic targets and strategies to modulate neuroinflammation, such as anti-inflammatory drugs, immune-modulating agents, and neuroprotective interventions. Attendees will gain valuable insights into the complex interplay between neuroinflammation and neurological disorders, deepening their understanding of the underlying pathophysiology. They will also explore the potential of novel therapeutic approaches that aim to regulate neuroinflammatory responses and prevent or slow down disease progression.

By attending this session, researchers, clinicians, and healthcare professionals will be equipped with the latest knowledge and advancements in neuroinflammation and its implications for neurological disorders. This session aims to foster collaborations, inspire innovative research, and ultimately contribute to the development of effective therapeutic strategies to combat neuroinflammation and improve patient outcomes in neurological disorders.